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anti ascc1  (Proteintech)


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    Proteintech anti ascc1
    Anti Ascc1, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 6 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/anti+ascc1/ASCC1+Antibody/pmc12216297-230-5-13
    Average 93 stars, based on 6 article reviews
    anti ascc1 - by Bioz Stars, 2026-09
    93/100 stars

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    Related Articles

    Software:

    Article Title: Pan-cancer analysis reveals ASCC family promotes the cancer progression of lung adenocarcinoma
    Article Snippet: .. The blots were treated with anti-ASCC1 (1:2000) (TA503721, Origene, China), anti-ASCC2 (1:500) (11529-1-AP, Proteintech, China), anti-ASCC3 (1:400) (17627-1-AP, Proteintech, China) and anti-GAPDH (1:5000) (ab181602, Abcam, UK) antibodies, the bands were visualized using enhanced chemiluminescence(AMERSHAM ImageQuant 800) and Adobe Photoshop 2021 software was used for image processing and assembly. ..

    Article Title: Pan-cancer analysis reveals ASCC family promotes the cancer progression of lung adenocarcinoma.
    Article Snippet: .. The blots were treated with anti-ASCC1 (1:2000) (TA503721, Origene, China), anti-ASCC2 (1:500) (11529-1-AP, Proteintech, China), anti-ASCC3 (1:400) (17627-1-AP, Proteintech, China) and anti-GAPDH (1:5000) (ab181602, Abcam, UK) antibodies, the bands were visualized using enhanced chemiluminescence(AMERSHAM ImageQuant 800) and Adobe Photoshop 2021 software was used for image processing and assembly. ..



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    Fig. 2 | Interactions of TRIP4 variants in cells. a Western blots (WB) monitoring immunoprecipitation (IP) of <t>ASCC1,</t> ASCC2, and ASCC3 by the indicated N-terminally Flag-tagged TRIP4 variants from the cell extracts. b Western blots (WB) monitoring immunoprecipitation (IP) of ASCC3 by the indicated HA-tagged
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    Image Search Results


    Primary antibodies used in this study

    Journal: Neural Regeneration Research

    Article Title: Mutual regulation of microglia and astrocytes after Gas6 inhibits spinal cord injury

    doi: 10.4103/NRR.NRR-D-23-01130

    Figure Lengend Snippet: Primary antibodies used in this study

    Article Snippet: ASCC1 , 1:200 , Origene , Rockville, MD, USA , TA503721S , AB_11127958.

    Techniques:

    Gas6/Axl signal pathway is inactive in reactive and scar-forming astrocyte after SCI. (A) The violin plot analyses were shown the change of the Gas6 and Axl expression in naive, reactive and scar-forming astrocytes clusters after SCI. (B) Immunofluorescence analysis of Ascc1 + (green, FITC) cells and Gas6 (red, Alexa Fluor 594) immunopositivity in the injured spinal cord. Ascc1 + cells and the immunopositivity of Gas6 were significantly decreased after SCI. (C) Immunofluorescence analysis of Nestin + (green, FITC) cells and Gas6 (red, Alexa Fluor 594) immunopositivity in the injured spinal cord. Nestin + cells were increased and the immunopositivity of Gas6 was significantly decreased after SCI. (D) Immunofluorescence analysis of Bcan + (green, FITC) cells and Gas6 (red, Alexa Fluor 594) immunopositivity in the injured spinal cord. Bcan + cells were increased and the immunopositivity of Gas6 was significantly decreased after SCI. (E) Immunofluorescence analysis of Ascc1 + (red, Alexa Fluor 594) cells and Axl (green, FITC) immunopositivity in the injured spinal cord. Ascc1 + cells and immunopositivity of Axl were significantly decreased after SCI. (F) Immunofluorescence analysis of Nestin + (red, Alexa Fluor 594) cells and Axl (green, FITC) immunopositivity in the injured spinal cord. Nestin + cells were increased and the immunopositivity of Axl was significantly decreased after SCI. (G) Immunofluorescence analysis of Bcan + (red, Alexa Fluor 594) cells and Axl (green, FITC) immunopositivity in the injured spinal cord. Bcan + cells were increased and the immunopositivity of Axl was significantly decreased after SCI. Scale bars: 100 μm (upper), 20 μm (lower). The data are presented as the means ± SD ( n = 3). ** P < 0.01, vs . sham group (unpaired t -test). A.U.: Arbitrary unit; Axl: AXL receptor tyrosine kinase; Bcan: brevican; DAPI: 4′,6-diamidino-2-phenylindole; FITC: fluorescein isothiocyanate isomer I; Gas6: growth arrest specific protein 6; SCI: spinal cord injury.

    Journal: Neural Regeneration Research

    Article Title: Mutual regulation of microglia and astrocytes after Gas6 inhibits spinal cord injury

    doi: 10.4103/NRR.NRR-D-23-01130

    Figure Lengend Snippet: Gas6/Axl signal pathway is inactive in reactive and scar-forming astrocyte after SCI. (A) The violin plot analyses were shown the change of the Gas6 and Axl expression in naive, reactive and scar-forming astrocytes clusters after SCI. (B) Immunofluorescence analysis of Ascc1 + (green, FITC) cells and Gas6 (red, Alexa Fluor 594) immunopositivity in the injured spinal cord. Ascc1 + cells and the immunopositivity of Gas6 were significantly decreased after SCI. (C) Immunofluorescence analysis of Nestin + (green, FITC) cells and Gas6 (red, Alexa Fluor 594) immunopositivity in the injured spinal cord. Nestin + cells were increased and the immunopositivity of Gas6 was significantly decreased after SCI. (D) Immunofluorescence analysis of Bcan + (green, FITC) cells and Gas6 (red, Alexa Fluor 594) immunopositivity in the injured spinal cord. Bcan + cells were increased and the immunopositivity of Gas6 was significantly decreased after SCI. (E) Immunofluorescence analysis of Ascc1 + (red, Alexa Fluor 594) cells and Axl (green, FITC) immunopositivity in the injured spinal cord. Ascc1 + cells and immunopositivity of Axl were significantly decreased after SCI. (F) Immunofluorescence analysis of Nestin + (red, Alexa Fluor 594) cells and Axl (green, FITC) immunopositivity in the injured spinal cord. Nestin + cells were increased and the immunopositivity of Axl was significantly decreased after SCI. (G) Immunofluorescence analysis of Bcan + (red, Alexa Fluor 594) cells and Axl (green, FITC) immunopositivity in the injured spinal cord. Bcan + cells were increased and the immunopositivity of Axl was significantly decreased after SCI. Scale bars: 100 μm (upper), 20 μm (lower). The data are presented as the means ± SD ( n = 3). ** P < 0.01, vs . sham group (unpaired t -test). A.U.: Arbitrary unit; Axl: AXL receptor tyrosine kinase; Bcan: brevican; DAPI: 4′,6-diamidino-2-phenylindole; FITC: fluorescein isothiocyanate isomer I; Gas6: growth arrest specific protein 6; SCI: spinal cord injury.

    Article Snippet: ASCC1 , 1:200 , Origene , Rockville, MD, USA , TA503721S , AB_11127958.

    Techniques: Expressing, Immunofluorescence

    Fig. 2 | Interactions of TRIP4 variants in cells. a Western blots (WB) monitoring immunoprecipitation (IP) of ASCC1, ASCC2, and ASCC3 by the indicated N-terminally Flag-tagged TRIP4 variants from the cell extracts. b Western blots (WB) monitoring immunoprecipitation (IP) of ASCC3 by the indicated HA-tagged

    Journal: Nature communications

    Article Title: Extended DNA threading through a dual-engine motor module of the activating signal co-integrator 1 complex.

    doi: 10.1038/s41467-023-37528-3

    Figure Lengend Snippet: Fig. 2 | Interactions of TRIP4 variants in cells. a Western blots (WB) monitoring immunoprecipitation (IP) of ASCC1, ASCC2, and ASCC3 by the indicated N-terminally Flag-tagged TRIP4 variants from the cell extracts. b Western blots (WB) monitoring immunoprecipitation (IP) of ASCC3 by the indicated HA-tagged

    Article Snippet: Western blotting was performed using antibodies against the Flag tag (Sigma-Aldrich F3165; 1:7500), ASCC1 (Proteintech 12301-1-AP; 1:500), ASCC2 (Proteintech 11529-1-AP; 1:1000) andASCC3 (Proteintech 17627- 1-AP; 1:1000).

    Techniques: Western Blot, Immunoprecipitation